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dc.contributor.authorCaverzan, Andréiapt_BR
dc.contributor.authorPassaia, Giselept_BR
dc.contributor.authorRosa, Sílvia Barcellospt_BR
dc.contributor.authorRibeiro, Carolina Wernerpt_BR
dc.contributor.authorLazzarotto, Fernandapt_BR
dc.contributor.authorMargis-Pinheiro, Márciapt_BR
dc.date.accessioned2014-02-28T01:50:54Zpt_BR
dc.date.issued2012pt_BR
dc.identifier.issn1415-4757pt_BR
dc.identifier.urihttp://hdl.handle.net/10183/87993pt_BR
dc.description.abstractWhen plants are exposed to stressful environmental conditions, the production of Reactive Oxygen Species (ROS) increases and can cause significant damage to the cells. Antioxidant defenses, which can detoxify ROS, are present in plants. A major hydrogen peroxide detoxifying system in plant cells is the ascorbate-glutathione cycle, in which, ascorbate peroxidase (APX) enzymes play a key role catalyzing the conversion of H2O2 into H2O, using ascorbate as a specific electron donor. Different APX isoforms are present in distinct subcellular compartments, such as chloroplasts, mitochondria, peroxisome, and cytosol. The expression ofAPX genes is regulated in response to biotic and abiotic stresses as well as during plant development. The APX responses are directly involved in the protection of plant cells against adverse environmental conditions. Furthermore, mutant plants APX genes showed alterations in growth, physiology and antioxidant metabolism revealing those enzymes involvement in the normal plant development.en
dc.format.mimetypeapplication/pdfpt_BR
dc.language.isoengpt_BR
dc.relation.ispartofGenetics and molecular biology. Vol. 35, n. 4 supl (Dec. 2012), p. 1011-1019pt_BR
dc.rightsOpen Accessen
dc.subjectEstresse abióticopt_BR
dc.subjectascorbate peroxidaseen
dc.subjectAntioxidantespt_BR
dc.subjectantioxidant systemen
dc.subjectreactive oxygen speciesen
dc.subjectPeroxidasept_BR
dc.subjectAscorbatopt_BR
dc.subjectabiotic stressen
dc.subjectmutant plantsen
dc.titlePlant responses to stresses : role ascorbate peroxidase in the antioxidant protectionpt_BR
dc.typeArtigo de periódicopt_BR
dc.identifier.nrb000876400pt_BR
dc.type.originNacionalpt_BR


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